Sunday, 4 August 2019

What we need to know about HCV Virus?


Thinking outside the BOX? HCV Antibodies to test or not to test??
–If we tets should it be routine or selective screening?
Q. 1A:- (antiHb) testing  for all or only in high risk cases :-Many do it as a routine basis. What is the reason? There is a standard guideline for "universal screening for any diseases globally". But those who doesn’t tets this as a routinely their argument is “Does this disease (antiHb) testing (HCV infection) can fulfill all the criteria-of universal screening?”..
HCV –is the  Test worthy?? If no specific low cost drug is available with high degree of efficacy-then guideline says that there is no rationality of universal screening.  But those who are in  favour of routine testing of (antiHb) testing  their argument is  it’s to take extra precautions for spillage and transmission to self and theatre/ L room staff.
The nuntold story of HCV:--What is the CV of HCV:- What is the similarity with HBV?? Ans:-Similar to HBV, fulminant lethal acute HCV infection can occur rarely. More typically, patients do not clear the virus on their own, so HCV develops into a chronic hepatitis and the patients become chronic carriers of the virus. Because many HCV infections are asymptomatic in the acute setting, the diagnosis is often made during the chronic phase. After exposure to HCV, usually parenteral, Hepatitis C antigen is present initially, and then antiHCV develops in the weeks following infection. The specific diagnosis of HCV infection requires serological and/or molecularbased (NAT) assays. In rare instances, antiHCV may not develop; in these cases, a definitive diagnosis of HCV requires molecularbased (NAT) assays. Spontaneous recovery from HCV infection occurs in 15–45% of infected individuals with recovery in the higher range occurring in women given antiD immunoglobulin contaminated with HCV. NAT of plasma intended for fractionation and of blood donations for HCV RNA began in the late 1990s when methodologies became available. NAT for HCV is now required in many countries. As antiHCV testing is also generally performed in addition to NAT, infected units from donors with spontaneous recovery from HCV would also be eliminated from the blood supply.

Q.1B:- (antiHb) testing: _ Issues that lurks in our mind: The issues on HCV: My task is to make you refresh on   the knowledge and initiating a debate.  HCV: Universal screening in India?? Or selective screening?-opinion of house??



Q.2:-When to implement selective screening??? Ans:-It is definitely indicated in women with H/o exposure to blood products &  all blood products, needle pricks, , IV drug abusers, H/O STD, Homosexuals, Such high risk women should always be screened prior to preg or in preg and if +ve adequate counseling be done

Q.3: Why worried about Virus?? Ans:-Infants may be affected if born to HbsAg or HCV Ab +ve mothers,

, Q.4:- What about preoperative testing?? Ans:-Prior to any planned surgery this is a must irrespective risk categorization-, major or minor.  Should be tested for (antiHb) testing, But unfortunately many avoids such useful trots in our country for reason best known to you.  
CV of HCV(contd) ::_Q.5:-: Is a drug available for cure of the disease or prevention of vertical transmission?? Ans:-No effective drug is available which is safe in preg?
Q.6: Why this virus is a bit little different in contrast to HbsAg?? Ans: - The good side of HCV is that in contrast to HBV fortunately the quantum of transmission by sexual route is less than that of HbsAg. Infectivity rate is less in than HbsAg.


Q7:-Fight goes on!!! Selective screening or Universal screening for HCV. Ans:-So far as I am concerned I feel Universal is better. We don’t know who will end up in CS or vacuum / Operative vaginal delivery / amniocentesis or CVS!! So the old idea of selective screening   does not hold well, Screened primarily during pregnancy for the benefit of healthcare professionals to prevent accidental injuries.

Q.8:-No treatment available for this virus:-But dilemma prevails:-Many are of opinion that HCV testing in ANC is just to add an extra cost burden on the family, more so as no management options is available if virus is present (tets is +ve), Sadly, till date no vaccination is available either in preg period if virus tets is -ve-in preg or post delivery.

CV of HCV(contd) Q.9: Then why testing: Own protection like HIV +ve, HbsAg ve cases?? But the fact remains there is a real threat of transmission to health care personal, we're achieving near 100% Institutional delivery-Normal/ instrumental. If +ve:-then advised universal standard precautions like HIV +ve, HbsAg +ve even if the patient is non infective. Infectivity rate is less in than HbsAg. Yet many of us are not doing routine screening as we do in case of Hep B. But there is strong demand and also all corporate hospitals are doing it routinely in elective cases and in few places for all antenatal cases. 



CV of HCV(contd) Transfusiontransmitted virus (TTV): Role of nucleic acid technology (NAT) testing?? Sadly, infections were not uncommon in the early days... Hepatitis B, hepatitis C and HIV transfusiontransmitted infections still do occur in the 21st century. In recent years with advanced technologies and improved donor screening, the risk of viral transfusion transmission has been markedly reduced but not Zero. . Hepatitis B virus (HBV), hepatitis C virus (HCV) and human immunodeficiency virus (HIV) have all shown marked reduction in transmission rates. However, the newer technologies, including nucleic acid technology (NAT) testing, have affected the residual rates differently for these virally transmitted diseases. Zero risk, which has been the goal, has yet to be achieved.
Q.14: Is it possible to have false –ve tets of such three transfusion related virus. Ans:-False negatives still persist, and transmissions of these viruses still occur, albeit rarely. It is known that HBV serological testing misses some infected units; likewise,

The story of HCV:: Silent killer in the form of poor quality of  life (hepatic subfuncion  for decades): Etiology of common cause of  Cirrhosis of Liver in 2019::_Not the alcohol, neither HBV infection persistence  but now HCV is coming fast as an imp cause of Hepatic failure as no antiviral agent neither vaccine is available, HCV induced cirrhosis:-Q.15: Surprisingly, HBV NAT–negative units of labeled blood or blood products have also been known to transmit the virus. Similarly, HIV miniboom NAT–negative units have transmitted HIV, as recently as 2007; likely, these transmissions would have been prevented with singleunit NAT testing.

 CV of HCV(contd) Q.17: Then what -: What is the full proof??

Ans: Use more new technologies, such as pathogen inactivation (PI), will (ideally) eliminate these falsely test negative components, regardless of the original testing method used for detecting the viruses.
Q. 19:-What is new in (antiHb) testing?
Nucleic acid technology (NAT) in 1999:-The first cases of transmission of a viral illness through blood transfusion were reported in 1943 .Laboratory testing for viral transfusiontransmitted viruses began in 1969 with testing for hepatitis B surface antigen (HbsAg). To reduce the risks of transmission of other viral illnesses (e.g. nonA, nonB viral hepatitis-now termed as Hep C), introduction of almandine aminotransferase (ALT) testing and antibody to hepatitis B core antigen (antiHb) testing began in the 1980s .More specific disease serological testing (antiHepatitis C virus antibody and HIV antigen and antibody testing) followed in the 1980s–1990s markedly reducing the risk of these transfusiontransmitted viruses (TTV).
Days [ 000 with a ‘window period’ (time from infection to first reactive test) of 30–38 000 to 1 in 357 .

What is NAT??  : -- Nucleic acid technology (NAT) was promulgated in 1999, Later there was further reduction in viral infections transmitted by blood transfusion. Using serological methods, the risk of Hepatitis B virus (HBV) is currently estimated to be 1 in 2823units of blood products in highest quality of blood banks...

Q.22 What about of the current risk of transfusiontransmitted Hepatitis C virus (HCV) infection with NAT testing in place along with serologic testing?? It is estimated to be 0·03–0·5 in 11million transfusions. Earlier it was million to 1 in 4·3. 

Q.23: What about HIV transmission?? For human immunodeficiency virus (HIV), transmission risk using NAT and serologic testing is currently estimated to be 1 in 154,. Although quite small today, the risks for transfusion transmission of HBV, HCV and HIV persist. So, efforts to reduce the risks to zero continue.
Hepatitis B
million  carriers of HBV worldwide, the infection, as measured by HBV surface antigen (HBsAg) positivity, remains endemically high in Africa, parts of Asia, the Middle East and parts of South America, ranging from 8% to 15% of those populations. In low prevalence areas, such as the United States, western and northern Europe, Canada and parts of South America, the prevalence is estimated to be after the discovery of widespread HBV infection became evident in 1963 with the introduction of HbsAg testing, HBV was noted to have a high prevalence in multiply transfused patients. However, it is now known that HBV is a very common infection in the general population and that transfusion transmission accounts for a minority of those infections. The prevalence of HBV differs by geography. With over 300 <2% [ 1]. Acute infection with HBV, whether transmitted via transfusion, intravenous drug use, perinatally or via sexual contact, may be asymptomatic or cause clinical viral hepatitis with nonspecific constitutional symptoms, including jaundice. Rarely, acute infections, including acute infections from transfusiontransmitted HBV, can cause culminant liver failure and death [6].
After transmission of HBV, HBV DNA is the first marker to be present in the blood. After viral replication in the liver, HBV viral load can attain 108–1010 viral particles per milliliters of serum [1]. Vermeil during the window period, however, is exponentially lower; infectivity, incidentally, in also lower in the WP, most likely related to antibodies (antiHb and antiHBs) formed after acute infection [7]. Testing for those infected with HBV historically involves detection of HbsAg. HbsAg is present in the serum of infected individuals from weeks to months after onset of infection and before symptoms begin. Some infected individuals never test positive for HbsAg, but generally will produce an antibody response to Hepatitis B core antigen (HbsAg): antiHBc. The fact that there are some falsenegative tests for HBsAg is the reason for testing for antiHBc in some countries. However, determination of HBsAgnegative/antiHBcpositive distant HBV infections in individuals, such as healthy blood donors without a history of hepatitis, testing falsely positive for antiHBc has been a persistent problem for blood donor collection facilities. Generally, two positive antiHBc tests will result in permanent deferral from donating blood in the United States. Recently, however, an algorithm for reentry of these donors has been approved by the US Food and Drug Administration (F.D.A.) [8].
Q.25:-NAT HBV DNA testing how effective??  How specific??  Unlike HCV and HIV testing, NAT HBV DNA testing has not eliminated the necessity for serological testing for HBV carrier donors. Where instituted, it was hoped that NAT testing for HBV would 1) reduce the window period for HBV, 2) identify lowlevel carriers of HBV, 3) provide another mechanism for reentry of HBsAg falsepositive donors and 4) ultimately replace serologic000 [ al testing. NAT testing for HBV DNA has been implemented to a variable extent in industrialized countries in Europe, North America, Asia, Australia and Africa. Occult HBV infections (HBsAg negative, HBV NAT positive) have been identified in all studies, in the range of 1/2000–1/1079. Japan was in the higher part of the range with the difference possibly being related to the use of larger pools for their NAT testing .
It has been shown that occult HBV infections can transmit the virus via blood transfusions, but the infectivity is not 100% [10], and the level of viremia needed to infect has not been determined in humans. However, in chimpanzees, it may be related to HBV genotype [11]. NAT testing for HBV varies by country and varies by blood centre within countries. HBV NAT testing is mandated in some countries [12]. Singleunit NAT testing can detect very low levels of HBV DNA (<IU/ml). With the availability of multiplex testing of small pools of donor sera, more blood centres are implementing HBV NAT, along with HCV and HIV. However, without singleunit NAT HBV DNA testing, the window period may not be shortened that much, compared to the sensitive tests available today for HBsAg (like PRISM/Abbott Park, IL, USA). This can be explained by the relatively slower doubling time of HBV in the window period, resulting in a lower viral load. Thus far the consensus is that NAT should be used in conjunction with serological testing to identify lowlevel infections as well as infections that are at the ends of the window periods of detection. There is no consensus either on whether singledonor NAT or what size pool NAT is optimal, and it may depend on the prevalence of HBV in the country. Additionally, HBV infectivity may depend upon the immunosuppressive state of the recipient of the blood component. 100 
Infectious disease testing on donated blood is of great importance in the reduction in risk for TT HBV infection. Other processes may also reduce the risk of HBV from blood components. For example, optimization of donor screening predonation is where most of the potential infected donors can be eliminated from the donor pool. This screening process could and should be individualized by country and even community. On the other end of the process, postdonation, pathogen inactivation (PI) may be almost 100% effective in eliminating the risk of HBV TTD from platelets and plasma in countries where it has been implemented. The role of effective testing remains critical, at this time, because of the lack of availability of PI for red blood cell components and whole blood. Finally, the role of vaccination for HBV also has the potential of almost completely eliminating the potential risk of HBV by blood transfusion; the reduction in risk that widespread vaccination can provide would be large in countries where the virus is endemic and access to vaccinations is currently limited.
Hepatitis C
After the discovery of hepatitis A virus (HAV) and HBV, a significant number of patients testing negative for HAV and HBV and with clinical presentation or laboratory evidence of viral hepatitis were identified; these patients were presumed infected with what was initially termed nonA, nonB (NANB) viral hepatitis.
HCN:- Most of these NANB viral hepatitis patients had a history of intravenous drug use. The majority of patients with NANB hepatitis later tested positive for HCV after testing for antiHCV became widespread in the 1990s. After testing was available, it was also verified that blood transfusion was another significant mode of transmission of HCV. The infection is present worldwide with lower incidence rates in North America, Australia and Europe (approximately 2%) and a higher rate in Egypt (>5%). 
Once infected with HCV, the virus travels via the blood to the liver where it replicates. Viremia occurs usually with few symptoms. Similar to HBV, fulminant lethal acute HCV infection can occur rarely. More typically, patients do not clear the virus on their own, so HCV develops into a chronic hepatitis and the patients become chronic carriers of the virus.
Because many HCV infections are asymptomatic in the acute setting, the diagnosis is often made during the chronic phase. After exposure to HCV, usually parenterally, Hepatitis C antigen is present initially, then antiHCV develops in the weeks following infection. The specific diagnosis of HCV infection requires serological and/or molecularbased (NAT) assays. In rare instances, antiHCV may not develop; in these cases, a definitive diagnosis of HCV requires molecularbased (NAT) assays. Spontaneous recovery from HCV infection occurs in 15–45% of infected individuals with recovery in the higher range occurring in women given antiD immunoglobulin contaminated with HCV. NAT of plasma intended for fractionation and of blood donations for HCV RNA began in the late 1990s when methodologies became available. NAT for HCV is now required in many countries. As antiHCV testing is also generally performed in addition to NAT, infected units from donors with spontaneous recovery from HCV would also be eliminated from the blood supply.
years of age [ 000 from 2005 to 2008. The increase has been primarily in Caucasian donors, especially in those donors over 50 000 to 2·98/100 Further reduction in transfusiontransmitted HCV, as well as any TTV, begins with improved donor screening. Recent data from the American Red Cross have shown that HCV positivity in blood donors has increased from a rate of 1·96/1004]. Donor screening, i.e. focusing donor questions on risk factors and on certain racial/age populations may aid in eliminating potentially infected donors prior to donation. A major risk factor identified in population screening is sharing of needles, especially in use of drugs, as well as reuse of nonsterilized needles. Having had surgery in a developing country (with unknown exposure to one of the blood) may be a risk factor as noted in a study from Mexico where risk factors to HCV infection was noted to be having had surgery or a prior blood transfusion [15].
It is known that there are presumed falsepositive HCV donors (antiHCV positive, HCV NAT negative). With further complex methods evaluating for immunological response to HCV (not ready for routine screening testing) [16-18], it is known that some of these donors are truly negative, some of those negative because of seasonal viral infections [19] and some of the donors being positive having recovered from the infection. However, it is not believed that these positive donations are capable of transmitting HCV. Unsafe endoscopy practices at a US endoscopic clinic may have exposed patients to viral infections [20]. In the absence of other risk factors, these patients would not have been identified as higher risk for HCV in donor screening.
days [ With the use of antiHCV antibody testing only, before HCV NAT testing, the risk of transfusion transmission of HCV was reduced considerably. The window period for HCV was approximately 701days [ ]. With the introduction of NAT, the window period has been reduced to approximately 12121]. NAT HCV testing is mandated in many industrialized countries. Combined antibody/antigen testing is available, but this combination still is not as sensitive as NAT [22]. Generally, pooled NAT testing identifies virtually all HCVinfected units because of the high levels of HCV RNA [2324]. In a South African study, singleunit testing was performed, and only one window period HCV NAT–positive unit was identified. It should be noted that South Africa has a low incidence of HCV. In a German study, multipool NAT was used (10–96 donations per pool), and only one documented case of transfusiontransmitted HCV was missed with multipool NAT over a 5year period. All three main genotypes were detected. Individual NAT testing may not improve upon the multipool testing; however, as noted in a case study, also from Germany, HCV was transmitted from a red cell unit that was negative by individual NAT HCV testing [21].
NAT testing has markedly improved the detection of HCV in donor units, as well as dramatically reduced the window period. However, the risk persists, albeit low. PI has been shown to virtually reduce the risk of HCV transmission to zero in vitro. This has great potential, especially in light of the fact that, unlike HBV, an effective vaccine against HCV is not available. PI is not currently universally available. PI would be of great benefit in developed countries where it could potentially reduce testing and the costs associated with testing. PI would be of greater utility in developing countries where universal testing is not available; testing for many infections would not be needed. The major limitation is that PI is only available for plasma and platelet products, and not for red blood cells. As noted in the justmentioned case study of individual NAT failure to identify a lowlevel case of infectious HCV, the component transmitting the HCV was a red blood cell unit.
HIV
In June 1981, the landmark report of five cases of Pneumocystis jiroveci (then P. carini) Pneumonia in otherwise healthy young men heralded a new disease eventually named acquired immune deficiency syndrome (AIDS) [25]. Shortly thereafter, AIDS was also reported in IV drug users and a few patients with Haemophilia. In December 1982, AIDS was diagnosed in a San Francisco baby who had received a platelet transfusion from a gay man who died of AIDS [26]. The realization that we had a new infectious agent (possibly a retrovirus) that had started to infiltrate the blood supply mobilized all blood collection agencies throughout the Western world to do everything possible to prevent potentially infected individuals from donating blood. The advent of AIDS also mobilized the research community to identify the causative agent and develop a test for it.
In the United States, the first donor deferral guidelines were put in effect in January 1983. Those early efforts to indefinitely defer prospective blood donors at risk and their sexual partners helped decrease the incidence of transfusionassociated AIDS by an estimated 90% [27]. The human immunodeficiency virus (HIV) (originally named HTLVIII) was described by Gallo and Montagnier [28] in 1984, and the first ELISA antibody test was available to blood banks in April, 1985. It is estimated that in the early 80s, the risk of being infected by a blood transfusion was in the order of 1:100 in some US cities, like San Francisco [27]. The focus, as well as the public perception, of transfusion medicine was changed forever.
days, respectively. The HIV P24 antigen test was introduced in March, 1996 with the hope of further reducing the window period. days. Second and third generation antiHIV tests reduced the window to 33 and 22 million people are infected. The first generation ELISA tests for antiHIV were able to detect the great majority of asymptomatic HIV carriers, but the window period (time to seroconversion) in newly infected individuals was approximately 56 million had died of AIDS. The vast majority of AIDS cases were not because of transfusions, however. In subSaharan Africa alone 24 million were newly infected and 2·8 million people were living with HIV world wide, that 4·1 In 2005, it was estimated by the WHO that 38
Transmission of HIV involving three different donors negative for both antiHIV and P24 antigen testing was documented in 1986 [29], 1987 [30] and 2007 [31months earlier tested positive with individual NAT, but had tested negative on a 96 sample minipool. Follo day one. The second one involved transmission of HIV by both platelets and RBCs. NAT on the undiluted donor plasma was positive, but detection was inconsistent in the 1:16 and 1:24 dilutions (the usual pool sizes in the USA). On the third case, a retained sample from a donation 4 day donation, but negative for the −11 days (double donation) prior to becoming HIV P24 antigen positive. Subsequent NAT testing was positive on the −4 ]. The first one was a frequent platelet donor who infected three recipients 11 and 4days. Minipool NAT testing had already been implemented in some European Countries (like Germany). wing this case, individual donation (ID) NAT testing for HIV, HCV and HBV was implemented in Denmark. Minipool NAT testing for HIV and HCV was introduced in US blood collection centres in 1999, reducing the HIV window of infectivity to 11
Five cases of HIV transmission by donations negative by NAT minipool testing have been reported in the United States from four donors [3233], one in Germany [5] and one in France [34]. In working up these transmissions in detail, there are several common findings: The viral load was too low to be detected by pooled testing which generally requires >copies/ml. By look back, it was determined that these donors’ seroconversions were recent, thus explaining the low viral load. More important, further questioning of the involved donors revealed risk factors, specifically recent male to male sexual contact, that were denied in the original screening interview. Retesting of stored donor samples with ID NAT was reactive in the cases where it was performed. 90 
The importance of viral load is further illustrated by a report by Ferreira and Nel [35copies/ml. days after collection; and fresh frozen plasma, originally sent for fractionation but retrieved for further testing. The viral load was estimated to be only 12 copies/ml. The sample tested negative for antiHIV and HIV p24 antigen. It tested positive, however, by NAT. The assay used has a sensitivity between 16·7 and 25 days after collection to a 20yearold patient; platelets, transfused as part of a pool to a 12year old patient 5 ]. In January 2003, a 52yearold male tested HIV antibody positive on his 55th blood donation to the South African National Blood Service. Look back on his earlier, HIVnegative donation from October 2002 showed that three components were made from it: RBCs, transfused 12
In January 2003, the recipient of the platelet pool tested positive for HIV antibody and HIV p24 antigen. Phylogenetic analysis of donor and recipient samples confirms the transmission from donor to recipient. The recipient of the red cells was tested for HIV antibody in March 2003 and found to be negative. Again in July 2003, he remained negative for HIV antibody, and NAT for RNA, the same test found positive in the platelet recipient.
000. Further analysis of the data revealed some disturbing trends: While in the years 2005/2006, 67 incident HIV positive tests (both NAT and serology) were found, in the years 2007/2008 there were 92, with an increase of 25 cases. The number of 16 to 19yearold males in both groups went from 6 to 21, a statistically significant increase ( 060 million donations tested, mostly pools of 16, there were 32 positive HIV NAT yield samples with an incidence of 1:2 years experience (1999–2008) after NAT minipool testing was implemented revealed the following: Of 66 In the United States, American Red Cross’ 10P <0·01). A preponderance of nonCaucasian donors was also noted [ 4]. These demographics parallel the incidence of HIV in the community. While there was no suggestion of test seeking behaviour when analysing the US data, a 2006 Brazilian study showed that donors still use the blood donation process to obtain HIV testing [36]. These finding underscore the importance of the initial donor history questionnaire, along with effective donor education, to ensure its veracity.
ID NAT testing is out of reach, as is pooled NAT testing, in many countries such as many in subSaharan Africa, where the incidence of HIV in the general population is expressed in double % digits. To significantly improve the safety of transfused blood, new technologies, such as PI, must be added to the already extensive battery of strategies [37]. Such systems have proven successful in preventing viral transmission when plasma and platelet components are treated [38]. They have been implemented in several European and Middle East countries, and FDA approval is being sought in the United States. So far, there is no available way to apply PI methods to red cells. The cost of such intervention has yet to be determined, but it will be undoubtedly quite high for the marginal increase in safety from HIV, HBV and HCV; but it may be worthwhile for emerging transfusion transmissible infections, especially for those agents for which we do not have effective tests or means to decrease their risks.
Less common viruses
Rarely, other identified viral infections have caused transfusiontransmitted infections. HAV is a foodborne illness that generally causes an acute clinical picture. As virtually all HAV infections are symptomatic, potential infectious donors would be eliminated by blood donor screening. There have been documented cases of transfusiontransmitted HAV [1], including remote cases involving transmission via contaminated factor VIII. HAV is not inactivated by solvent/detergent treatment, but HAV vaccines are available. Hepatitis E virus (HEV) infection, like HAV, is another enterovirus generally caused by fecally contaminated water. The presentation of HEV infection is similar to HAV, but subclinical infections are reported. Transfusiontransmitted HEV is rare, but has been reported. Antibody testing for HEV infection is generally used to test for infection, but PCR testing is available. A recent study from Japan showed the prevalence of antiHEV in qualified blood donors to be 3·4% [39], slightly higher than that reported in the United States and Europe [1]. Like HAV, solventdetergent treatment is not effective. Theoretically, PI would inactivate HEV as well as HAV.
Conclusion
million for HIV. For maximum reduction in risk, testing of units may not be able to reduce the rates of infection further. The best opportunity for near zero risk is treatment of the blood components directly, such as with PI. Although not approved worldwide, this technology has been shown to reduce the rate to near zero with the potential of eliminating extensive testing of all units [ 000 for HBV to 1 in 2 Careful donor screening has been shown to eliminate the vast majority of HBV, HCV and HIV transfusiontransmitted viral infections over the past four decades. With viral specific antigen and antibody testing, viraltransmitted infections have been reduced another log. With NAT testing over the past decade, the risk of transmitting these infections through transfusion has been reduced further in the range of 1 in 30040]. PI has not been fully adopted and needs further investigation into the quantifying of pathogen reduction and toxicity. It has been suggested that, in addition to infectious disease testing which has reduced window period and established infections, PI could reduce transfusiontransmitted viral infections by reducing preNAT window period and occult infections [41]. Clinical studies proving this have not yet been performed.
As PI does not rely on specific disease testing, it allows elimination of risk for yet unknown emerging infections. Furthermore, PI has been shown not to affect protein composition or clotting capacity [42]. A major limitation on this technology, however, is that is not available for use in red blood cells, although it has been suggested that treatment of whole blood before fractionation could assist in resolving this issue [43-45]. The challenge persists to develop a method capable of achieving the near zero risk of TTV from red blood cells. Careful donor selection remains the best way to prevent TTV before the unit is even collected and tested.
References






The Society for Maternal-Fetal Medicine (SMFM) has issued guidelines for screening and management of Hepatitis C infection in pregnancy.  

The guidelines were published ahead of print in American Journal of Obstetrics and Gynecology.

Prevalence of HCV (Earlier teemed as NONE A, Non B hepatitis):-:-It is estimated that on average nearly 1-2.5% of pregnant women in US are infected with the virus and the risk of vertical transmission is 5%.

The recommendations of “The Society for Maternal-Fetal Medicine (SMFM”) are:

 Why all of us need to brush up our knowledge on Hepatitis C virus? Tips & Tricks on the virus: Hepatitis C:-1) firstly It is a RNA virus, & surprisingly this virus is the commonest cause of Post transfusion hepatitis!!! - . 2) set principles for “population based screening”:-do not apply to this particular virus as such screening tests are a) not cost effective b) serological tests are not highly sensitive, & highly specific but HCV RNA PCR is considered as most effective, c) no acceptable treatment course if diagnosed to be affected, though for the rich men & women α-interferon, & Ribavirin is reasonably effective for coupe of yrs (these are not possibly recommended in pregnancy period). 3) No recognized drug protocol as to “how best to prevent transmission to partner”., “partners treatment-though there is ongoing debate whether this virus is at all transmitted by sexual intercourse or not.-most believe that the rate of transmission is of little significance.

Then to summarize if one do not follow routine screening then Who must  be screened irrespective of her financial constraints ? Who are at risk of acquiring HCV infection?? It is indicated in women with H/o exposure to blood products &all blood products should be tested for HCV. Only for I. V. Drug abusers, patients &staff involved in dialysis programmers, pts with HIV /HBV infection, partners of pts with HIV/HBV/HCV , with h/o body piercing /tattooing, pts with unexplained elevated transaminases, in if from donors, recipients of organ transplants. No intervention can be done to prevent mother to child transmission. Hep Virus C: - The good news is that a) transmission to partner is life long relationship is < 5%.-so why barriers?? It is expected after one person is affected (mostly by transfusions) will live hopefully a health life for about 20-30 yrs of symptom free active life.


 Dr Nidhi Goel: Listen to the  bad news on HCV: - : - 1) Significant risk of (60-70%) OF DEVELOPING Char infection-body therefore poorly clear the virus and 20% of such Char carriers develop cirrhosis liver by 10-25 yrs time.2) the prevalence in India is about 1% (I am not very sure about current statistics); COUPLE OF YEAR BACK the global burden was > 300 million!!
2) There is no proven intervention to cut down vertical transmission-but the good news is that “mother-to-child transmission” is only possible when viral load is > 104 copies per ml of serum!! This level is seldom achieved.

HCV prevalence slightly less than HbsAg. But much more than HIV in a study on blood donors in blood bank.
he hepatitis C virus (HCV) infects some 170 million people worldwide--more than four times as many as HIV--and is causing rising rates of liver disease. Like HIV, it is a wily foe for researchers developing drugs and vaccines, because HCV also mutates rapidly, creating a swarm of different viruses in each infected person that can thwart antibodies easily. But progress is beginning to be made: Although to date no one has been able to grow HCV reliably in a laboratory culture of cells, on page 110, researchers describe a new system for culturing HCV's RNA. And a study on page107 offers one possible explanation for why some HCV strains are more resistant to interferon, the standard treatment for HCV, than others.

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Testing of individual blood donations for HCV RNA reduces the residual risk of transfusiontransmitted HCV infection








First published: 24 April 2002


Cited by: 28
BACKGROUND: To allow costeffective RNA testing with NAT techniques, the national authorities of several countries have planned or already introduced tests of mixed specimens, that is, plasma pools.
STUDY DESIGN AND METHODS: Highthroughput extraction, amplification, and detection of HCV RNA from individual blood donations were optimized and validated. The feasibility of the method and the frequency of antiHCVnegative, HCV RNApositive donations were determined in a prospective study of 27,745 allogeneic and 792 autologous individual donations.
RESULTS: The 50 and 95percent detection limits of the method were determined at 44 IU per mL and 162 IU per mL, respectively (World Health Organization HCV reference material).

When 201 HCV RNApositive sera were taken as a reference, the sensitivity was 97.5 percent. The assay specificity was determined at 99.77 percent.

During a 20month period, two seronegative blood donors tested positive in HCV PCR. The viral load of these donations was 6 × 106 and 3 × 107 copies per mL, respectively. Thus, the yield of HCV RNA testing in this study was 7.63 per 100,000 screened donations (95% CI, 1.2522.07). In both PCRpositive donors, seroconversion was found in subsequent blood samples.
CONCLUSION: This study compares the feasibility of singledonation HCV RNA screening, with the detection of a relatively high percentage of windowphase donations, to data reported from groups using HCV RNA testing of plasma pools. The relative yield of NAT of individual donations versus minipools should be directly investigated in the near future.


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 The naked truth in Blood Banks at night time in rural areas:-The virus  is transmitted mostly by parenteral route like HBV, less through sexual route.  Most infections, when probed revealed  in retrospect that infection was transmitted  is by BLOD TR more so before 1995 & still today  in our country-.For instance in Mouffasil (District level-Talks) Blood Banks where the screening of donated blood is not rigorously screened for all the 6 common pathogens by Lab personnel specially at night time..
We are undone at some emergent situations!! But what options we have in cases of PPH?? We are undone. , But in myoma patients TAH we can preoperatively improve Hob level née IV Fe therapy FCM etc thereby delaying planned surgery. So far world prevalence is considered about 1% of world population are affected by HCV. HBV is DNA but HCV is RNA virus, more vertical transmission in HBV is not so much in HCV. Sexual TR of HCV is questionable, but vertical transmission rate of HBV is more common in comparison to HCV Ab titer is often remain unchanged    after the storm of HCV which is not so much with HBV. The prevalence of HBV infection about 2-6% in adults as Char carriers where as in case of HCV is 1%only.  For HCV it takes about 10 yrs to have S/S of char hepatitis( anorexia, loss f wt diarrhea etc), ,it takes 20 yrs to develop cirrhosis as exemplified by USF and loss of venous pulsation of Portal vein with dilated PV in spleen portal Doppler.
It takes about 30 yrs to develop hepatocellar Ca. In those residual damages is much more with HCV than HBV. All women who are at increased risk for Hepatitis C should get tested for anti-HCV antibodies at their first prenatal visit. If the risk persists till later in pregnancy or new risk factor arises, the screening should be repeated again.

Who are more prone to have infection?? According to American Liver Foundation the risk of Hepatitis C is increased /may be: acquire 
·                     Shared needles to inject drugs or straws to inhale them
·                     Had tattoos or body piercings in an unclean environment using unsterile equipment
·                     Worked in a place where one can  came in contact with infected blood or needles, for example, healthcare workers
·                     Received a blood transfusion or organ transplant
·                     Received a blood product for clotting problems
·                     Needed to have your blood filtered by a machine (hem dialysis) for a long period of time because your kidneys weren’t working
·                     If someone is born to a mother with HCV
·                     Had unprotected sex with multiple partners
·                     Have or had a sexually transmitted disease
·                     Have HIV
What else to be screened??   A) ELISA test is 95% sensitive but HCV RNA virus is the most sensitive test and considered as gold standard. All HCV positive pregnant women should be screened for other sexually transmitted diseases (STDs) including HIV, syphilis, gonorrhea, Chlamydia, and hepatitis B virus (HBV). In acute cases perinatal TR (vertical) is 0-18%. . Immunoglobulin to neonate may be administered after birth 0.5 ml and Rpt after 4weeks.After acute inn of HCV as high as 80% will develop Char liver diseases. 35% will dev Cirrhosis & 5% will develop Hepatocellar Ca,
Transmission rate varies with HCV RNA titers: - For instance if RNA titer is less than 104 copies per ml3 then chance of transmission to foetus is almost Zero. By contrast if titer is 106 than chance if vertical TR is about 36%!!! ) All patients with HCV, including pregnant women should be advised to refrain from consuming alcohol-hepatotoxicity may be accelerated / augmented...

4) Is there any drug (effective against HCV) antiviral agent available in India Direct-acting antiviral medications (DAA) like Ribavirin & Interferon are contraindicated, therefore not to be used in pregnancy, except in settings of clinical trials? The treatment should be initiated after the delivery of the patient in the postpartum period.
5) If invasive prenatal testing is requested than amniocentesis is preferred over chorionic villus sampling (CVS) because of limited data on the risk of vertical transmission for CVS over amniocentesis. Similarly scalp b monitoring in labour and prolonged labour is avoided... Mode of delivery does not affect the risk of transmission to foetus.
 6) HCV infection solely is not an indication for Cesarean section in absence of other obstetric indication.

7) Internal fetal monitoring, prolonged rupture of membranes, and episiotomy should be avoided during labor in HCV+ women
8) Risk of transmission to foetus depends on maternal HCV RNA titers , If titer load is < 10 4 copies per ml3 then minimal risk, But  if  such figure is > 106 copies per ml then neonatal transmission rate will be somewhere 36%!!!!But those mothers who have HIV as well in such cases the rich of transition will be as highs 19% irrespective of maternal RNA leas,

8) Positive HCV status is not a contraindication for breast feeding. 








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