Monday, 17 February 2020

ABC of oestrogens


1)                      .ABC OF Oestrogens : A refresher course for members working at periphery:-Types of Oestrogens in the Indian market: A) Ethinyl Oestradiol –mostly as COC B) Oestradiol Valerate-mostly available as oral Tablets e.g. Trade names are E D Val 2; Evadilol, 3) Equine estrogens-safe-e.g. Premarin 4) Natural Oestrogens-Abbott.  Fortunately .Diethyl stilboesterol, like Quinestrol  is also   not available in Indian market
2)          While prescribing oestrogen alone or in conjunction with Progesterone –do we afford time to select the best suitable friendly but effective  oestrogen for that woman and for that diseases for whichthe drug is prescribed ? Let’s have of a look on types of Oestrogens available in Indian Trade: Let us be acquanatied who is who! What oestrogen for what disease So far in descending potency, the oestrogens used in trade can be arranged in flowing order:1) EE(Ethinyl  Estradiol)-most  potent  -Lynoral Tablet 0.01 mg / 0.05 mg 2) Oestradiol –used in subfertility for improving ET as oral Tablets . Used as Valerate salt. Actually E2 is 17-beta estradiol which stands in between very active pharmacological potent EE and CEE(less potent).  3) CEE- Premarin Tab-commonly used for secondary amenorrhoea. 4) Oestriol (E3)-Evalon. Oestriol oral / vaginal form is most innocuous and safe for months /Years.
Efficacy of different estrogens in body varies from one woman to other: The reasoning of one gene to one protein synthesis does  not hold good in cases of oestrogens as density receptor and down regulation / up regulation of cell kinetics varies.;-The differences between the various products lie in the specific estrogenic substances they contain.
Oral oestrogens even in identical dose, in some cases, they affect one part of the body more than another. For the most part, however, estrogen products are interchangeable, as long as differences in dosage are taken into account.
Enzymes in human Endometrium, Local production of bioactive estrogens  & correlation of ET with serum oestrasiol :  “The understanding of  Thickness of Endomatrium”:- Endometrium  contain variety of hormone-producing  enzymes and coenzymes . Activity of endometrial enzymes varies in diff women even amongst sisters. Such enzymes are 1) Sulphatase  which converts inactive oestrone  to active estrone ; 2) aromatase and 3) 17 delta- OH steroid dehydrogenase (HSD).These last two enzymes   cause conversion of all types of androgens coming to endometrium via local circulation  e.g. Androstenedione, T4 to bioactive  oestradiol.
 That is one reason why PCOS women (better termed as Androgen Excess disorders)à do exhibit thick ET almost all times.
Therefore the thickness of Endo does not always correspond with the serum E2. Even if stimulated cycles oral replacement of E. Valerate for thin ET on day 19-12 at the dose of 8mg OD schedule fail to improve ET. Even in IVF settings inspite of high dose of Gonadotrophins ET remains poor thereby compelling the specialists for Freeze à than Transfer in subsequent cycles. Therefore Ladies and gentlemen –ET  depends partly on the activity of such enzymes, their potency , in addition to E2 Receptor population of estrogens (up regulation/ down regulation)/ !!
Locally produced Oesradiol, therefore,   play a major role in thickness of Edo.--> May be one of the cause of AUB.
 Researchers have documented that Progesterone given systemically or as LNG-IUS does case diminution of all three enzymes quoted above thereby inactivate the quantum of local synthesis of estradiol and estrone. Progesterone therapy therefore à cause availability of less bioactive estrogens,
2) What are the routes of administration at our disposal?? Preparations & Route of adminisarion of oestrogen either as immunotherapy or with Progesterone? - 1) Oral route-mostly, 2) Vaginal route as creams-pessary, 3) Transdermal 4) Transdermal System)5)  IM –Inj Progynova.& Combined E & P as contraceptive On monthly basis. What is the most common indication of consumption of oestrogens? Indications of Oestrogens as a single agent or as COC with Progesterone?? As oral contraceptives B) treatment of DUB C) Tr of PMS D) Delaying the dates of period –social reasons E) Dysmenorrhoea –not responding to analgesics noncontraceptive benefits of Oral contraceptives. F) Endometriosis-Tricycling particularly.
The rare uses of COC: - Hirsutism, Pretreatment before IVF/IUI to decrease the level exaggerated LH/ Androgens. Osteoporosis, abnormal bleeding of the uterus, vaginal irritation, female castration, and Turners syndrome. Estrogens may also be prescribed for birth control. They are effective as a "morning after" contraceptive but should only be used as an emergency treatment because of the damage they can cause to developing fetuses.
Limitations of Oestrogen therapy? There is no evidence that these drugs are effective for nervous symptoms or depression occurring during menopause: They should not be used to treat these conditions; they should be used only to replace the estrogen that is naturally absent after menopause.
Types of oestrogens: Though total six types of estrogens are available -only 3 are actually present in significant amounts: estrone, estradiol, and estriol. E-1 Estrone is the most potent of the three and is the major estrogen produced by the ovaries. E-2 17beta Estradiol is the most prevalent and is naturally modified to estrone, which is then turned into E-3 estriol, the least potent of the three. All of the Estrogens listed in this section will produce equal effects and side effects when their doses are equal, taking their various potencies into account. More potent medicines require a smaller dose to produce the same effect. E-2 is the most prevalent estrogen produced by the ovaries prior to menopause.



 COC related possible Health Risks.
Myocardial Infarction:-...2) Cerebral Ischemic Stroke. 3) Cerebral hemorrhagic Stroke. 4)  Deep venous Thrombosis.  5)  Pulmonary Embolism. ?6) Breast Cancer.  ?7) Cervical Cancer.
development of blood-clotting disorders, liver cancer or other liver tumors, high blood pressure, glucose intolerance (symptoms similar to diabetes) or worsening of the disease in diabetic patients, unusual sensitivity to the sun, and high blood levels of calcium.


Side effects of Drug Vis- a- vis Health risks induced by the drug consumes: Known health risks of oestrogens if used for months together: At the outset we must be clear that side effects of a drug should not be confused with health risks of the client (patient-who is consume sing the drug/ device somebody is using) --either for illness or for prophylaxis. Of concern is particularly metabolic and coagulatory side effect of oestrogens. Whenever we are using oestrogens for 6 months and beyond these questions should lurk in our mind.  We have to take into consideration about side effects of estrogens Vis-a -Vis desired benefits out of the drug. Needless to mention that we always calculate the “Risk/Benefit ratio” in such a setting. Vaginal estrogen creams may stimulate bleeding of the uterus. They can also cause breast tenderness. Vaginal discharge, and withdrawal bleeding (if the product is suddenly stopped). Women with endometriosis may experience heavy vaginsl bleeding.
Top of Form
Side effects of COC:-The most common side effects include nausea, vomiting, breakthrough vaginal bleeding, vaginal spotting, changes in menstrual flow,  no menstrual period during and after estrogen use enlargement or tenderness of the breasts, swelling of the ankles and legs, loss of appetite, weight changes, retention of water, abdominal cramps, and feeling of bloatedness. The estrogen patch can cause skin rash, irritation, and redness at the patch site.
Less common side effects are, , enlargement of uterine fibroids, vaginal infection with Candida, a cystitis-like syndrome, , loss of scalp hair, and development of new hairy areas. Lesions of the eye and contact-lens intolerance have also been associated with estrogen therapy. Rarely one may have may experience migraine headache, mild dizziness, depression, and increased sex drive (women) .
Rare side effects include stroke, blood-clot formation, dribbling or sudden passage of urine, loss of coordination, chest pains, leg pains, difficulty breathing, slurred speech, and vision changes. Men who receive large estrogen doses as part of the treatment for prostate cancer are at a greater risk for heart attack, phlebitis, and blood clots in the lungs.Estriol (E3) is metabolically / innocuous so also from the standpoint of coagulator parameters. We always do Minimal/ No Hypercoagulable sates.E3-Estriol is of lest potent and mostly used for local application –Cream/Ointments --either as prophylaxis/ to prevent urogenital atrophy or to prevent senile vaginnits/ recurrent urethritis in postmenopausal women. Occasionally this Oestriol is used along with Probiotics-as vaginal pessary in idiopathic BV (Bacterial Vaginosis) in resistant cases where Metranidazole, Clinidamycin have failed.
We , busy practitioners often forget the Relative contraindications of estrogens alone/ COC . Honestly speaking, some of us do prescribe COC/ Oestrogen unnoticing:-e.g.1) HTN 2) Smokers 3) > 10 yrs COC use continuously. 4) Originally –Normotensive but with the use of COC/ E2-there is gradual rise of BP 5) Dyslipidaemia. We often forget to limit the duration of therapy in such cases .This is rush at OPD or at Clinic. As a result some women if gain benefit out of COCà  continue to take COC for long time without further consulation  which is to be discouraged. Women, especially those over 35, who smoke cigarettes and take an Estrogen have a much greater chance of developing stroke, hardening of the arteries, or blood clots in the lungs. The risk increases as age and tobacco use increase

Absolute contraindications: 1) DM with retinal/ CAD who are on Insulin 2)CRF  3) had P/H/O VTE 4) Gross/Moribund  Obesity 5) Had Heart attack 6) On Tr for Ca Cx / Breast Ca.

 Newer & safer Oestrogens in oral Pills----Widening the horizon of choice of Contraception: Can we come out of synthetic oestrogencontING pills?? Such Pills (COC containg Ethinyl Oestradiol) may be avoided in contraceptive selection should we not go for Women friendly / metabolic friendly Oestrogens whole choosing Pills for Indian women??
  Your views please:-We are aware that synthetic oestrogens (EE-ie. Ethinyl oestradiol) is globally the most commonly used estrogen   in COC preparations.
But this type of Oestrogen salt (EE) is metabolically a bit harmful and should not be used for long time specially  in women with some medical diseases like DM, those with exaggerated risk of VTE, hypertension, hepatic diseases , ages> 40 yrs, dyslipidaemia, & smoker. Put in such a situation, earlier the contraceptive seeker was deprived of the benefit of Oral pills. Instead such medically sick women were offered to choose TCu-380A or Tubectomy or even the least effective Barrier methods.   Now the fact that more safe oestrogen i.e. Estradiol valerate is being used in some countries as estrogen component in COC (though it is costly) in lieu of synthetic EE -we can offer such sick women valerate containing COC or even  more new COCs –( COC with absolutely Natural Oestrogens   )--which are metabolically more safe than  valerate containg COC.
This policy if adopted by us and financially supported by Govt. (subsidy) then popularity of COC may be doubled than the present rate of COC use in our country... There may not be any incidence of heart attack/ cerebral thrombosis/ venous thrombosis in women with the use of newer oestrogen containg COC. Many women are afraid of taking COC for the fear of such dreaded complication which they have read in media/prints. Such safer oestrogen containg pills are more relevant in women who are already at increased risk of Heart attack or DM prevails.
 We can now safely prescribe to such at risk women Valerate containg COC.Now about the widespread use of Oestrogen Valerate: The following are the: - What are the currently licensed products used as OCP with natural Oestrogens abroad?
A) Pills where natural oestrogen used is Oestradiol Valerate are at UK= Qlaira, Klaira (this product is originally developed by Teva and later Merck (originally Organon) also had an agreement with Teva about marketing sratetegy, 
At USA= Natazia (Bayer
). In such formulations the progesterone used is Dienogest.
One should remember that E. Valerate is a prodrug and is very quickly converted to natural oestrogen (E2).
B) Pills containing Plain Oestradiol (E2): No salt at all= Zoely (MSD). In such formulation the progesterone used is nomegestrol which is commonly used in contraceptive implants.

-10) Differential Proliferation of one type of cells where many types of tissues are existent:-
11) Anybody has tried Bazedoxifene? It is being used abroad with the approval of FDA in combination with Conj Estrogen (CEE-Conj Equine Estrogen).
Oestrogens & VTE: -- Estrogen cause slight increase in fibrinogen and by that promotes slightly of the contraceptive steroids we should remember that it is the estrogen which causes Hypercoagulable state and is primarily attributed for DVT and allied thrombosis during OCP intake. The risk of VTE is directly related to dose of E but the OCP induced risk of VTE IN PREGNANCY IS LOWER THAN THE RSIK ASSOCIATED WIRH EVEN LOW-DOSE COC.
Inherited Thrombophilia:-  But as we know in some countries factor Leiden mutation, Protein-C /S synthesis disorders (DEFICIENT PRODUCTION) or prothrombin mutation disorders are to the extent of 0.5 to 5% of general population in those countries. In such countries it will be prudent to screen women who candidates for inherited thrombophilias and refrain from prescribing OCP if screen +ve or family is +ve/ or she herself has already suffered from DVT in the recent past... This is not true for our country.

Acquired APC resistance: - APC resistance: - APC naturally down regulates the thrombin formation. APC therefore is a naturally occurring the prevalence of DVT in acquired APC resistance is 6/10,000 women in reproductive years. Who is not.OCP? But if one uses OVP then the prevalence goes up to Increased APC resistance can invite thrombosis.
PCOS & Venous thrombosis:- using Thrombosis can occur at varying sites including legs, thigh veins, lungs, eye ,intestines or heart
Natural estrogens with Dienogest are QLARIA
Natural estrogens with Dienogest are QLARIA
E2 valerate with Dienogest as progesterone as OCP:-Brand names are:-Klaira, Natazia. –all quadriphasic
D). Progesterone Receptor Modulators
The side effects of Visanne (dienogest-Bayer) are Chloasma, depression, irregular bleeding, ovarian cyst formation. Wt Gain, Nausea.  . Will 1 mg dose (anovulatory doe) will be able to manage endometriosis. 

Will combination of Dienogest with natural   Oestrogen be at all effective to control endometriosis, keeping in mind that the very diagnosis has not been confirmed in this case? The idea of prescribing Dienogest containing natural oestrogens stems from the fact such drugs will ensure monthly bleeds. I understand many gynaecologists will purposefully intend to avoid EE (synthetic agent) which is very potent and will exert more harmful effect on the existing pathology (endometriosis).
Such combinations of natural oestrogens with Dienogest are QLARIA –polyphasic preparations (marketed again by Bayer) and the Obstr. Component used is E2V (valerate). The other trade names are Klaira, Natazia. –all quadriphasic.

I appeal some ART specialist to consider for trial on couple of cases particularly in women where fertility is not an issue (therefore no question of loosing time to conception – post Tubectomy endometriosis).    Bayer Co. / Endometriosis Society of India/. FOGSI can help in funding if may be approached.
What about other Progesterone Receptor Modulators in this young adult?   Will any Indian Doctor become team leader in study of ULIPRISTAL ACETATE (UPA) in Pel? Endometriosis.
Dienogest. Brand Names are: - Visanne (Dienogest-Bayer).

Ulipristal as ECP. EllaOne.
AS ECP:-  It is an FDA approved drug for Emergency contraceptive used as 30 mg single dose as post coital contraceptive (Morning after pill). Research has confirmed that ULIPRISTAL ACETATE (UPA) is more effective that LNG 1.5 mg. The trade name is EllaOne. –

Realtive Contraindications of minipils:--Regarding the apprehension raised by members about the use of  minipills progesterones( though WHO has referred to LNG and not specially to Dienogest) or even COC in a case impaired hepatic function (fatty liver/ NAFL- non-alcoholic fatty liver) :---- may I draw attention to the fact that WHO medical Eligibility criteria put use of LNG as category  2 risk  only in cases with 1) Non vascular DM    2) DM with nephropathy      3) P/H/O preg Cholestasis (cat 1 risk-no risk)/ past COC related Cholestasis(Cat 2 risk    4)  Acute viral hepatitis  ! (Not to speak of Chr carriers)  -believe me I am quoting from original WHO BOOK, Ed. 2010, ISBN 978 92 4 15388 8) p. 53       5) Valvular heart disease     ) Multiple risk factors for arterial CVS disease (!)  7) Controlled HTN with drugs. ) no age bar 9) Smokers of all ages (!). 10) Obese women 110 DVT on oral anticoagulants (!) & Myomas.
Such views have also been expressed by other academic bodies like Clinical Guidance. Contraception for women aged over 40 yrs by UK, Australia and USA separately.
 I am not very sure whether such liberal use will be safe for dienogest too. 3) Another issue- The compulsion of use of a drug for treating disuse as using dienogest cannot be equated to contraceptive prophylaxis where other options are available.
Again ICMR and its control Six population research centers, “Human Reproductive Research centers” = Total such centers are 31 in India,  20 ICMR study centers can forward to ace o pilot study on this issue. ENDOETRIOSIS is a nightmare to ART specialists!!!

There are several Types of oestrogen used in Pharma industry:- A) EE(Ethinyl  Estradiol) –most potent I call it handle with care so far as VTE is considered in unfavourable alterations in coagulatory proteins synthesized  from liver B) CEE-e.g. Manufactured  from Pregnant Mare Urine: Trade name   Premarin- available in strengths 1.25 mg, 0.625 mg, & 0.03 mg. Tablets of the lowest  strength (0.03 mg )  ,however are most commonly used along with MPA(Medroxy progesterone-2.5 mg / even 5 mg  / Duphaston either as sequential or continuous combined  as it is commonly called  HRT for months/ years with minimal monitoring / harm to the women concerned .In fact CEE is less potent than EE(EE=Ethinyl Oesradiol) which, as I said, is  commonly used in COC/COCP. I repeat by saying EE is very potent. C) Oestradiol:-17 β oestradiol is Natural Estrogen secreted from Ovaries, Adrenal and Adipose tissues. The issue of HRT & choice of oes COC related Health Risks.
Myocardial Infarction:-Cerebral Ischaemic Stroke.
Cerebral hemorrhagic Stroke.Deep venous Thrombosis.Pulmonary Embolism.Breast Cancer.Cervical Cancer.
Oestrogen: Dose & duration: - Sex hormones are implicated in the immune response, with estrogens as enhancers at least of the humoral immunity and androgens and progesterone (and glucocorticoids) as natural immune-suppressors. Several physiological, pathological, and therapeutic conditions may change the serum estrogen milieu and/or peripheral conversion rate, including the menstrual cycle, pregnancy, postpartum period, menopause, being elderly, chronic stress, altered circadian rhythms, inflammatory cytokines, and use of corticosteroids, oral contraceptives, and steroid hormonal replacements, inducing altered androgen/estrogen ratios and related effects. In particular, cortisol and melatonin circadian rhythms are altered, at least in rheumatoid arthritis (RA), and partially involve sex hormone circadian synthesis and levels as well. Abnormal regulation of aromatase activity (i.e., increased activity) by inflammatory cytokine production (i.e., TNF-alpha, IL-1, and IL-6) may partially explain the abnormalities of peripheral estrogen synthesis in RA (i.e., increased availability of 17-beta estradiol and possible metabolites in synovial fluids) and in systemic lupus erythematosus, as well as the altered serum sex-hormone levels and ratio (i.e., decreased androgens and DHEAS). In the synovial fluids of RA patients, the increased estrogen concentration is observed in both sexes and is more specifically characterized by the hydroxylated forms, in particular 16alpha-hydroxyestrone, which is a mitogenic and cell proliferative endogenous hormone. Local effects of sex hormones in autoimmune rheumatic diseases seem to consist mainly in modulation of cell proliferation and cytokine production (i.e., TNF-alpha, Il-1, and IL-12). In this respect, it is interesting that male patients with RA seem to profit more from anti-TNFalpha strategies than do female patients.

Spontaneous miscarriage and preterm delivery are common complications of pregnancy. Pro-inflammatory cytokines have been shown to be associated with recurrent spontaneous miscarriage (RSM) and preterm delivery (PTD) and these have led to exploration of ways to down regulate pro-inflammatory cytokines and/or to upregulate anti-inflammatory cytokines. Progesterone-induced blocking factor (PIBF) is a molecule with inhibitory effects on cell-mediated immune reactions. We have ascertained the effects of PIBF on secretion of selected type 1 and type 2 cytokines by peripheral blood mononuclear cells from healthy non-pregnant women, women undergoing normal pregnancy, women with unexplained RSM and women with PTD. Peripheral blood mononuclear cells from 30 women with a history of unexplained RSM, 18 women undergoing PTD, 11 women with normal pregnancy and 13 non-pregnant healthy women were stimulated with a mitogen in the absence and presence of PIBF after which the levels of cytokines released into culture supernatants were determined by ELISA. Production of the type 2 cytokines IL-4, IL-6 and IL-10 by lymphocytes from the RSM and PTD groups and of IL-4 and IL-10 by lymphocytes from healthy pregnant women was significantly increased upon exposure to PIBF, while the levels of type 1 cytokines were not affected. Ratios of type 1:type 2 cytokines were decreased, suggesting a shift towards Th2 bias. PIBF did not affect cytokine production by lymphocytes from non-pregnant women. Thus, PIBF acts on lymphocytes in pregnancy to induce a type 1 to type 2 cytokine shift by upregulating the production of type 2 cytokines.
PMID:

19371956

[PubMed - indexed for MEDLINE] Good/Ill effects of oestrogen varies: The metabolism of oestrogens in body varies from one women to other, from one group of population to other (Ref: Goldzieher, JW, Am J Obstet & Gynaecol 163:318,1990). There is even a range of variability at different sampling times within the same individual. Therefore it is not surprising that the same dose can cause side effects in one individual (Vaginal adenosis, Neoplasia, testicular cancer as stated by members) and not in others.(source: ‘A Clinical Guide  for Contraception’, 4th Ed, by Leon Speroff, Philip D Darney. Lippincott, pp; 31)

 Now about the widespread use of Oestrogen Valerate: = May I mention the followings: - What are the currently licensed products used as OCP with natural Oestrogens abroad?
Pills where natural oestrogen used is Oestradiol Valerate= B are at UK= Klaira, Klaira (this product is originally developed by Teva and later Merck (originally Organon) also had an agreement with Teva about marketing sratetegy,
At USA= Natazia (Bayer). In such formulations the progesterone used is Dienogest.
One should remember that E. Valerate is a prod rug and is very quickly converted to natural oestrogen (E2).
Pills containing Oestradiol (E2) = Zoely (MSD). In such formulation the progesterone used is nomegestrol which is commonly used in contraceptive implants. By the way if one tries to avoid Parenteral Oestrogens what are other types of natural oestrogen? Which can be considered in lieu of Inj. Oestradiol Val. Thanks?





Good/Ill effects of estrogen vary: The metabolism of oestrogens in body varies from one woman to other, from one group of population to other (Ref: Goldzieher, JW, Am J Obstet & Gynaecol 163:318, 1990). There is even a range of variability at different sampling times within the same individual. Therefore it is not surprising that the same dose can cause side effects in one individual (Vaginal adenosis, Neoplasia, testicular cancer as stated by members) and not in others. (Source: ‘A Clinical Guide for Contraception’, 4th Ed, by Leon Speroff, Philip D Darney. Lippincott, pp; 31)

Can synthetic oestrogens be avoided in COC products?? We are aware that synthetic oestrogens    are bit harmful and should not be used for long time in women with some medical diseases like DM, those with exaggerated risk of VTE, hypertension, Dyslipidaemia. In such a situation, earlier the contraceptive seeker was debited of the benefit of Oral pills. Instead they were asked to choose TCu-380A or Tubectomy.   Estradiol valerate is used in lieu of synthetic EE in COC.Now about the widespread use of Oestrogen Valerate: The following are the: - What are the currently licensed products used as OCP with natural Oestrogens abroad?
A) Pills where natural oestrogen used is
Oestradiol Valerate are at UK= Qlaira, Klaira (this product is originally developed by Teva and later Merck (originally Organon) also had an agreement with Teva about marketing sratetegy, 
At USA= Natazia (Bayer
). In such formulations the progesterone used is Dienogest.
One should remember that E. Valerate is a prodrug and is very quickly converted to natural oestrogen (E2).
B)
Pills containing Oestradiol (E2) = Zoely (MSD). In such formulation the progesterone used is nomegestrol which is commonly used in contraceptive implants. By the way if one tries to avoid Parenteral Oestrogens what are other types of natural oestogen?

May I draw attention to all concerned that

Natural estrogens with Dienogest are QLARIA
1.                       Natural estrogens with Dienogest are QLARIA
E2 valerate with Dienogest as progesterone as OCP:-Brand names are:-Klaira, Natazia. –all quadriphasic
D). Progesterone Receptor Modulators
The side effects of Visanne (dienogest-Bayer) are Chloasma, depression, irregular bleeding, ovarian cyst formation. Wt Gain, Nausea.  . Will 1 mg dose (anovulatory doe) will be able to manage endometriosis. 

Will combination of Dienogest with natural   Oestrogen be at all effective to control endometriosis, keeping in mind that the very diagnosis has not been confirmed in this case? The idea of prescribing Dienogest containing natural oestrogens stems from the fact such drugs will ensure monthly bleeds. I understand many gynaecologists will purposefully intend to avoid EE (synthetic agent) which is very potent and will exert more harmful effect on the existing pathology (endometriosis).
Such combinations of natural oestrogens with Dienogest are QLARIA –polyphasic preparations (marketed again by Bayer) and the Oestr. component used is E2V (valerate). The other trade names are Klaira, Natazia. –all quadriphasic.

I appeal some ART specialist to consider for trial on couple of cases particularly in women where fertility is not an issue (therefore no question of loosing time to conception – post Tubectomy endometriosis).    Bayer Co. / Endometriosis Society of India/. FOGSI can help in funding if may be approached.
What about other Progesterone Receptor Modulators in this young adult?   Will any Indian Doctor become team leader in study of ULIPRISTAL ACETATE (UPA) in Pel? Endometriosis.
1.                       Dienogest. Brand Names are: - Visanne (Dienogest-Bayer).

Ulipristal as ECP. EllaOne.
AS ECP:-  It is an FDA approved drug for Emergency contraceptive used as 30 mg single dose as post coital contraceptive (Morning after pill). Research has confirmed that ULIPRISTAL ACETATE (UPA) is more effective that LNG 1.5 mg. The trade name is EllaOne.

Realtive Contraindicatins of mnipils:--Regarding the apprehension raised by members about the use of  minipills progesterones( though WHO has referred to LNG and not specially to Dienogest) or even COC in a case impaired hepatic function (fatty liver/ NAFL- non-alcoholic fatty liver) :---- may I draw attention to the fact that WHO medical Eligibility criteria put use of LNG as category  2 risk  only in cases with 1) Non vascular DM    2) DM with nephropathy      3) P/H/O preg Cholestasis (cat 1 risk-no risk)/ past COC related Cholestasis(Cat 2 risk    4)  Acute viral hepatitis  ! (Not to speak of Chr carriers)  -believe me I am quoting from original WHO BOOK, Ed. 2010, ISBN 978 92 4 15388 8) p. 53       5) Valvular heart disease     ) Multiple risk factors for arterial CVS disease (!)  7) Controlled HTN with drugs. ) no age bar 9) Smokers of all ages (!). 10) Obese women 110 DVT on oral anticoagulants (!) & Myomas.
Such views have also been expressed by other academic bodies like Clinical Guidance. Contraception for women aged over 40 yrs by UK, Australia and USA separately.
 I am not very sure whether such liberal use will be safe for dienogest too. 3) Another issue- The compulsion of use of a drug for treating disuse as using dienogest cannot be equated to contraceptive prophylaxis where other options are available.
Again ICMR and its control Six population research centers, “Human Reproductive Research centers” = Total such centers are 31 in India,  20 ICMR study centers can forward to ace o pilot study on this issue. ENDOETRIOSIS is a nightmare to ART specialists!!!



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