Wednesday, 18 September 2019

Drug induced foetal adamge : Teratogensis


 When an embryo is most vulnerable to be attacked by a teratogens?? Risk of teratogenscity Mother is far away : This is the time there is no savior to young kid(blastocyst)  and blastocyst is vulnerable / prone to be attacked by drugs/ bad environment .  When blastocyst  is vulnerable to any attack by drug??  Dose and duration of drug counts!! How early at what dose of drug??  : When mother is far away!!!! During the first 3 days after ovulation development takes place in the fallopian tube( I call it mother is away!!-not nearby to protect)  . At the time of fertilization a pronuclear stage exists during which the nuclei from the egg and the sperm retain their integrity within the egg cytoplasm. After the pronuclear fuse the fertilized egg begins a series of mitotic cell divisions. The two cell stage is reached about 30 hours after fertilization. With continued division the cells develop into a solid ball of cells, which reaches the endometrial cavity about 3 days after fertilization. Thereafter a fluid filled cavity forms within the cell ass at which time the conceptus is called a blastocyst , The number of cells increases from about 12 to 32 by the end of the third day to 250 by the sixth day.
Until about 3 days after conception any cell is to potential that is capable of initiating development of any organ system. For this reason separation of cells during this period gives rise to monozygotic twins usually each normal . At the blastocyst stage cells first begin to differentiate. By this stage o development the embryo is located in the uterus where implantation occurs 6 to 7 days after conception.
One group of cells forms the inner cell mass that will ultimately develop into the fetus. Different tissues arise from each of the three cell layers. Brain nerves and skin develop from the ectoderm the lining of the digestive tract, respiratory tract and part of the bladder as well as the liver and pancreas from the endoderm and connective tissue cartilage muscle blood vessels the heart kidneys and gonads develop from the mesoderm . The   group of cells forming the periphery of the blastocyst is termed the trophoblast. The placenta and the fetal membranes develop from this outer cell layer.
Drug 12:- More information about Alcohol : Each year between 1,000 and 6,000 babies in the United States are born with fetal alcohol syndrome (FAS) . This is pattern of mental and physical birth defects that is common in babies of mothers who drink heavily during pregnancy. Even moderate or light drinking during pregnancy may harm the baby Anticoagulants Drug 13:- Low –molecular weight heparins may have substantial benefits over standard unfractionated heparin. The molecules are still relatively large and do not cross the placenta . The half life is longer allowing for once daily administration. However enoxaparin is cleared more rapidly during pregnancy so twice daily dosing is a advised. Low molecular weight heparins have a much more predictable dose response relationship obviating the need for monitoring of partial thromboplastin time . There is less risk for heparin induced thrombocytopenia and clinical bleeding at delivery but studies suggesting less risk for osteoporosis are preliminary.
The risk of heparin during pregnancy may not be justified in patients with only a single remote episode of thrombosis in the past. Certainly conservative measures should be recommended such as elastic stockings and avoidance of prolonged sitting or standings.

Drug 14  : Warfarin L: heparin have been  discus cussed  : Warfarin has been associated with chondrodysplasia punctata.  Which is similar t the genetically determined conradi-Hunermann syndrome. Warfarin embryopathy occurs in about 5% of exposed pregnancies and includes nasalhypoplasia  bone stippling seen on radiologic  examination ophthalmologic abnormalities including bilateral optic atrophy and mental retardation . Ophthalmologic abnormalities and mental retardation may occurs even with use only beyond the first trimester. The risk for pregnancy complications is higher when the mean daily dose of warfarin is more than 5 mg. Women with mechanical heart valves especially the first generation valves require warfarin anticoagulation because heparin is not safe or effective, Heparin treatment is associated with more thromboembolic complications and more bleeding complications than warfarin therapy.

Drugs: 15:-Thyroid and Antithyroid Drugs :Prophylthiouracil and methimazole  both cross the placenta and may cause some degree of fetal goiter. In contrast the thyroid hormones triiodothyronine and thyroxine cross the placenta poorly so fetal hypothyroidism produced by antithyroid drugs cannot be corrected satisfactorily by administration of thyroid hormone to the mother. Thus the goal of such therapy during pregnancy is to keep the mother slightly hyperthyroid to minimize fetal drug exposure. By the third trimester 3.0% of women no longer need antithyroid medication.
However in 2009 the FDA released a black box warning highlighting serious liver injury with PTU treatment to a greater extent than methimazole. Some authors are now advocating treatment with PTU only during the first trimester and switching to methimazole for the remainder of the pregnancy.
The need for thyroxin increases in many women with primary hypothyroidism when they are pregnant as reflected by an increase in serum thyroid stimulating hormone concentrations. Because hypothyroidism in pregnancy may adversely affect the fetus possibly by increasing prematurity it is prudent to monitor thyroid function throughout pregnancy and to adjust the thyroid dose to maintain a normal TSH level. It is recommended that women with hypothyroidism increase their levothyroxine dose by about 30% as soon as pregnancy is confirmed and then have dosing adjustments based on TSH levels. 

Take home message on drugs & teratogenic by Drugs : Illegal drugs, including cocaine, marijuana and Ecstasy, may cause birth defects and must be stopped if a women contemplates a pregnancy, medical help may be required to help her .Infections & cog abnormality  : Certain infections can cause birth defects when a woman gets them during pregnancy. About 30,000 babies a year (about 1 in 150 newborns) in the US are born with a viral infection called cytomegalovirus (CMV) . About 8,000 infected babies each year develop permanent disabilities, including mental retardation and loss of vision and hearing . Pregnant women often get CMV from young children who have few or no symptoms. Sexually transmitted infections (STIs) : Untreated syphilis can cause stillbirth, newborn death or bone defects

  Biology of embryo & embryopathy: What the obstetricians need to know: we have to play safe Five weeks after conception the embryo first begins to assume features of human appearance. The face is recognizable with the formation of discernible eyes nose and ears. Limbs emerge from protruding buds digits cartilage and muscles develop . The cerebral hemispheres begin to fill the drain area and the optic stalk becomes apparent. Nerve connections are established between the retina and the brain. The digestive tract rotates from its prior tubular structure and the liver starts to produces blood cells and bile. Two tubes emerge from the pharynx to become bronchi  and the lungs have lobes and bronchioles. The heart is beating at 5 weeks and is almost completely developed by 8 weeks after conception. The diaphragm begins to divide the heart and lungs from the abdominal cavity . The kidneys approach their final form at this time. The urogenital and rectal passages separate and germ cells migrate toward the genital ridges for future transformation into ovaries or testes. Differentiation of internal ducts begins with persistence of either mullerian or Wolffian embryos. The embryo increases from about 6 to 33 mm in length and increases 50 times in weight.
Three things to consider before U sign the order of “HANG THE  EMBRYO TILL DEATH” !! A hard task indeed!!!  : Fight by artillery/Nave./ by Air force?? Is the arms of stone age (Timing of Exposure), Dose of drug . & duration exposed and obviously what drug was prescribed-Was theta two agents alcohol& antiepileptics operating on a single woman  w]backed up elderly husband with sperms exhibiting much telomere damage , increased DNA Fragmentation?? All these  need to be addressed before offering a sc judgment. A hard task indeed to kill an imbrue-hang the embryo to death and sign the handing order hay prescribing Mifepristone !!!!
The second principle is that susceptibility of the conceptus to teratogenic agents varies with the developmental stage at the time exposure. This concept of critical stages of developmental is particularly applicable to alterations in structure. It is during the second to the eighth weeks of development after conception – the embryonic period – that most structural defects occur. For such defects it is believed that there is a critical stage in the developmental process after which abnormal embryogenesis cannot be initiated. For example neural tube defects result from the failure of the neural tubes to close. Given that this process occurs between 22 and 28 days post conception any exogenous effect on development must be present at o before this time. The neural tube has five distinct closure sites that may respond differentially to agents and may respond differently in timing. Investigation of thalidomide teratogenicity have clearly shown that effects of the drug differ as function of the developmental stage at which the pregnant woman took it.
The fourth principle is irrespective of the specific deleterious agent the final manifestations of abnormal development are death malformation growth restriction and functional disorder. The manifestation is thought to depend largely on the stage of development at which exposure occurs; a teratogen may have one effect if exposure occurs during embryogenesis and another if the exposure is during the fetal period. Embryonic exposure is likely to lead to structural abnormalities or embryonic death; fetal exposure is likely to lead to functional deficits or growth restriction.
Despite the importance of teratogen timing on specificity of anomalies a general pattern usually emerges with respect to any given teratogen. This will be evident throughout this chapter as we consider various agents. If no pattern is evident for a purported teratogen, it increases the suspicion that any purported association is spurious the observation reflecting confounding variables not recognize and hence not taken into account.
Which drug?? (The issue of time of ingestion , dose & for what period –for how many days?)
The fifth principle is that access of adverse environmental influences to developing tissues depends on the nature of the influence . This principle relates to such pharmacologic factors as maternal metabolism and placental passage. Although most clearly understood for chemical agents or drugs the principle also applies to physical agents such as radiation or heat. For an adverse effect to occur an agent must reach the conceptus either transmitted indirectly through maternal tissues or directly traversing the maternal body.
At what does the offending agent was prescribed?? Dose Effect(The issue of time of ingestion , dose & for what period –for how many days?)
The final principle is that manifestations of abnormal development increase in degree from the no-effect level to the lethal level as dosage increases. This means that the response may be expected to vary according to the dose duration or amount of exposure. For most human teratogens, this dose response relationship is not clearly understood but along with the principle of critical stages of development these concepts are important in supporting causal inferences about human reproductive hazards. Data regarding in utero exposure to ionizing radiation clearly show the importance of dose on observed effects for teratogens has been noted

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