Acute PID :
What are the sonological characteristics?? What
are the soologicalsigns of acute endometritis??
Ans: Besides , 1) free fluid
,2) hydrosalpinx 3) oophoritis. 4) There
may be thickened irregular endometrial
myometrial junction which can be used a strong supportive sign for
inflammatory reaction. 5) Thickened endometrium
alone however can’t be used as the diagnostic criteria for acute inflammation more so in such increased
thickness is observed in preovulatory
and secretory phases which is
normally present. Acute endometritis cause
to 6) edema à edema includes the endometrium and also the subendometrial
tissue due to its close anatomical
vicinity and 7) hyperemia due to neoangiogenesis..Dilated
blood vessels are seen in subendometrial
layer to because in this layer harbour the
dilated spiral vessels ( the main feeding vessels of the endometrial vessels 7 endometrial tissues 8) Endometrial
oedema:- Extravasations of the
fluid from these dilated vessels may also be
the cause of edema in the
subendometrial layer. 9) Why irregular endo-myometrial zone like adenomyosis??
Ans:-
Moderate
oedema leads to irregularly
increased thickness
of the endometrial myometrial
junctional zone . Endometrial
edema may appear as
increased thickness of the endometrium with a homogeneous texture.
It may have a ground galss like
echogenicity and both because of the echogenicity similar
to that of the normal myometrium and also because of irregularity
of the JZ and may be difficult
to distinguish from the
myometrium. There may eb fluid
in the endometrial cavity that is turbid.
How helpful is USG in diagnosisg acute PID/ endometritis??
What about Doppler findings on acute endometritis? A) Low resistance blood flow, which signifies
neoangiogensis(new blood vessels formation) in the endometrium is common in early secretary
phase . This is so because for last 3
decades WE KNOW THAT that normally during
the preovulatory and secretory
phase - there is physiological neo angiogenesis in the endometrium:-. . In
acute PID , too there is new blood vessels formation. Therefore this process of
neoangiogensis as identified by Doppler cannot always be used as diagnostic criteria for diagnosis of acute inflammatory process. Therefore this sign
can be used only in a phase of the cycle
when normally the endometrium is
avascular or has high resistance blood
flow. This phase is early
proliferative phase. Doppler must be used for
assessment of the vascularity. The neoangiogenesis of inflammation is
documented on ultrasound as 1) increased
vascularity in the endometrium in which vessels are typically have larger diameter
than the normal spiral vessels
and 2) low resistance blood flow. 3) Besides, the vascular
distribution may be heterogeneous due
to nonuniform involvement of the
endometrium in the inflammatory process. B) How to measure blood flow in
endometrium?? Ans:-The 3D ultrasound is useful for assessment of
the coronal
plane and is a modality with proved
ability for assessment of the endometrial myometrial
junction. C) How to measure ET?? Tips:-endometrial thickness is measured from outer margin of the hyperechoic line to the outer
margin of the hyperechoic line at the broadest part of the endometrium perpendicular to the central line of the endometrium.
How
many members are performing transrectal scan which according to few sonologist is preferred to
transabdominal route USG of endometrium?? :-The endometrium is thin and at
times ill defined and may also at times
contain fluid in the cavity. Endometrium is best evaluated by transvaginal
scan but a prior abdominal scan is recommended
for evaluation of the pelvic
anatomy. In cases when transvaginal scan is not possible or
denied by the patient transrectal scan is preferred to
transabdominal route.
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